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it is selectively recruited to the impaired mitochondria under stress conditions where it mediates phagocytosis and subsequent degradation of the mitochondria (Koyano et al., 2019)

The neuroprotective effects of limiting infarct and improving neurological function by semaglutide are dose-dependent against tMCAO in non-diabetic rats and are at least as strong as liraglutide (114)

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