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Targeting ALDH16A1 mediated thioredoxin lysosomal degradation to enhance ferroptosis susceptibility in SMARCA4-deficient NSCLC

Glucagon-like peptide 1 receptor stimulation reverses key deficits in distinct rodent models of Parkinson's disease

In addition, the project was also funded by the Albert Phlsson Foundation, the Swedish Diabetes Foundation, ALF, the Novo Nordisk Foundation, the Royal Physiographical Society in Lund, and the Medical Faculty at Lund University

Exceeding common ranges without strong scientific justification is generally discouraged in responsible research