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Rather, it is to engineer a pharmacologic signal in which glucagons catabolic properties are preserved while its hyperglycemic liability is buffered by incretin-mediated glucose control

Missing one application should not drastically affect results

[1] [2] A 2011 Cochrane review showed a HbA1c reduction of 0.20% more with exenatide 2 mg compared to insulin glargine, exenatide 10 g twice daily, sitagliptin and pioglitazone

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