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Melatonin administration reversed this phenotype by adopting a compensatory mechanism aimed at increasing Bcl-2 protein expression, blocking Bax proapoptotic activity via the SIRT1/NF-kB axis with a consequent and significant inhibition of Cyt C release and the lack of apoptosome formation and caspase 3 activation 37 (Fig

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Purine metabolism was involved in the NLRP3 inflammatory pathway and the Cyclic adenosine monophosphate-protein kinase A (cAMP-PKA) pathway [64], which might account for its influence on CAG

Wendel A, Fausel M, Safayhi H et al (1984) A novel biologically active seleno-organic compoundII

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