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It also fuels nucleotide biosynthesis and sustains oxidative phosphorylation in mitochondria

While eliminating such library species is not always possible, reducing the frequency of mispaired species within the library is critical to ensuring its phenotypic fidelity

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Accumulation of lagging events immediately after PH and throughout the progression in G1, including the alteration of the finely tuned activations of JNK and ERK1/2, considerably delays the commitment to DNA replication and mitosis