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BPC 157 not only increased VEGFR2 expression in vascular endothelial cells several hours after treatment but also rapidly induced VEGFR2 internalization within minutes, subsequently activating the phosphorylation of VEGFR2, Akt, and the eNOS signaling pathway, independent of known ligands or shear stress [93]

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This relationship supports the hypothesis that much of tirzepatide's anti-inflammatory benefit may be mediated through weight loss rather than direct receptor-mediated anti-inflammatory signaling, though controlled studies specifically addressing this question are limited