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This has an important impact on overall metabolism and energy balance, making AMPK a target for the treatment of UC

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Transcriptomic analysis revealed that VCPIP1-driven AMPK1-K234 deubiquitination inactivates AMPK and disturbs cardiomyocyte mitochondrial respiration, while VCPIP1 deficiency rescues hyperglycemia-induced mitochondrial dysfunction

R.OldfieldM.SchmidtA