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The enhancement of RaTG13- and Khosta-2 S protein-mediated cellcell fusion following post-overlay elastase treatment suggests that elastase may function not only as a priming protease that converts the spike into a fusion-competent form, but also as a triggering protease that cleaves the spike after ACE2 engagement to directly initiate membrane fusion (Fig

Insurance still may decide not to cover a medication, even if FDA indications for prescribing that medication are met, says Johnson-Rabbett

To enhance the detection of minor gene effects, future studies could focus only upon middle cerebral artery stroke

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