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Inhibition of mTOR with agents such as rapamycin would be expected to switch immune cell metabolism from glycolysis to oxidative phosphorylation and therefore may be detrimental to effector T cells but beneficial to regulatory T cells and macrophages in a Th1 or M1 state ( 7 Mitochondrial quality control and inflammation Mitochondrial quality control provides essential homeostatic functions including the regulation of the mitochondrial population to meet cellular energy demands, as well as the disposal of damaged mitochondria ( 7.1 Role of autophagy in mitochondrial quality control Mitochondria are the major source of energy in the form of adenosine triphosphate to fuel cellular processes

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